Life Extension Magazine®

Green tea leaves that may mitigate a number of diseases

Green Tea Reduces Mortality

Studies confirm the ability of green tea consumption to mitigate the risk of dying from any cause. In response to higher intake of green tea and its extracts, research reveals a reduced risk of developing cancer, diabetes, and vascular and neurodegenerative disorders.

Scientifically reviewed by: Dr. Shaylind Benson, ND, in August 2023. Written by: Martin Stein.

Green tea is considered one of the healthiest drinks on the planet.1

Besides its ability to improve circulation, it can help stabilize blood sugar and combat underlying causes of cancer.2-6

But it’s not just that green tea is healthy—it can reduce your risk of dying!

A decade of meticulous research evaluated hundreds of thousands of people in relationship to their dietary habits. The results showed that the higher the consumption of green tea and its polyphenol constituents, the lower the overall risk of dying from any cause. This longevity benefit is especially pronounced in older populations.1,7

Green tea combats many underlying processes that contribute to premature aging and disease.

Here, we’ll evaluate compelling studies showing that green tea reduces all-cause mortality. Then we’ll look at how green tea or its components help reduce the impact of specific health problems that contribute to premature death.

Green Tea Reduces Mortality

Green Tea Reduces Mortality  

For many years, green tea and its compounds have demonstrated compelling benefits in basic science and animal studies. But until about a decade ago, there were conflicting results about those health benefits in people.1,8

Now, a handful of epidemiological cohort studies has made the human health benefits much clearer.

In these types of studies, researchers follow a large group of people over a long period of time, recording both the health outcome of interest (death from all causes) and ingestion of substances (green tea consumption) that might influence the outcomes. Once the data have been collected, they are analyzed in search of statistically significant associations between ingested substance and the outcome.

By early 2017, several large cohort studies and meta-analyses of smaller cohort studies had been completed examining the association between green tea consumption and death from all causes. While the results varied somewhat by design and by the populations being studied, they all had one thing in common: They found significant reductions in all-cause mortality among habitual green tea users, compared with nonusers or low-level consumers.1,7-11

The largest individual studies involved populations of 40,000 to more than 160,000 subjects, and they all showed significant all-cause mortality risk reductions of 11%-18% in those drinking the largest daily amounts of green tea (five or more cups), compared to non-green tea drinkers.1,7-9 Those studies also found that the more tea a person drinks, the greater the risk reduction.

A smaller but still sizable study of 14,001 older adults found a still more impressive overall risk reduction of 58% in the highest-consumption group—a figure that rose to 68% in women only.10

Finally, a meta-analysis pooling the results of 18 individual cohort studies found a 20% reduction in all-cause mortality risk among the highest vs. the lowest category of green tea consumption.11 That study also broke down the data to show that for each one-cup increment of green tea consumed daily, the risk of all-cause mortality fell by 4%, a figure that supports the overall reductions in the preceding studies.11

Together, these studies vividly illustrate the powerful ability of green tea consumption to mitigate the risk of dying from any cause.

Green Tea Fights Age-Related Diseases

How is green tea consumption capable of producing such drops in the risk of death across the board? The reason is because green tea possesses multi-targeted properties that combat five fundamental processes that underlie degenerative disease: oxidative stress, sugar-induced damage (glycation), inflammation, mitochondrial dysfunction, and disruptions to the intestinal microbiome.12-20

As a result, green tea consumption has been found to have a beneficial impact on the chronic conditions responsible for killing the majority of older adults, including cardiovascular and neurodegenerative diseases, diabetes/obesity, and cancer, as well as some less-obvious factors like dental problems. Let’s look at each individually.

Cardiovascular Disease


Cardiovascular disease remains the leading killer of older Americans and is therefore a major contributor to all-cause mortality. Green tea consumption is widely recognized as a means of lowering cardiovascular disease risk.

Each of the studies mentioned earlier that showed significant reductions in all-cause mortality risk also showed significant decreases in the risk of dying from cardiovascular disease. Specifically, those who consumed the largest amounts of green tea reduced their risk of dying from cardiovascular disease by 14% to as much as 76% (and up to 82% for women).1,7-11

These benefits are largely driven by green tea’s high content of catechins (a type of polyphenol), the most abundant of which is epigallocatechin gallate (EGCG).21-23

Higher levels of green tea catechins are associated with lower levels of triglycerides and LDL cholesterol.22 EGCG has potent protective effects against the oxidation of LDL cholesterol.21-23 This is important because oxidized LDL cholesterol is a major risk factor for, and early contributor to, atherosclerosis and plaque formation that blocks arterial blood flow.23

In vitro studies demonstrate that green tea’s effects can protect against oxidized LDL-induced endothelial dysfunction (an early finding in atherosclerosis) by modulating the production of two forms of nitric oxide that regulate signaling changes in blood pressure and flow.23


Diabesity (the combination of type II diabetes and obesity) is another major contributor to premature death because it sets up older adults for metabolic, cardiac, neurologic, and malignant catastrophes by feeding inflammatory changes throughout the body.

Green tea—and especially the EGCG contained in green tea extracts—powerfully fights many of the causes and consequences of diabesity.

A human study revealed that taking 90 mg/day of EGCG increases the body’s ability to burn fat for energy, which results in increased energy expenditure. These actions not only combat the accumulation of fat, but also help prevent inflammation caused by fat cells.24 An animal study showed that EGCG can reduce body weight and shrink fat mass, in large part by stimulating the burning of fat for energy.25

Other favorable effects of green tea extracts and EGCG include reductions in fat uptake and liver fat storage, reductions in markers of fat-induced chemical stress, reductions in fat-induced insulin production, and reductions in inflammation produced by fat cells.26 As an added benefit, green tea extract prevents fat absorption by inhibiting the pancreatic enzymes required for its digestion in the intestine.27

What You Need to Know
Green Tea

Green Tea Reduces Mortality

  • A recent barrage of studies has confirmed that people who consume green tea are significantly less likely to die from all causes put together.
  • The risk of dying may be reduced by as little as 5% and as much as 76%, depending on study design and population, but the results are consistent across multiple studies.1,7-11
  • Green tea and its extracts exert this remarkable anti-mortality effect by reducing the risk of developing fatal chronic diseases, including cardiovascular and neurodegenerative diseases, obesity and diabetes, and cancer, as well as other conditions that predispose us to an early death, like tooth and gum disease.
  • Research demonstrates that the green tea components, EGCG and others, exert these widespread effects because of very specific and focused impact on a small handful of biochemical and cellular processes.
  • Green tea extracts should be considered an essential part of an overall strategy to extend life and evade disease.

Neurodegenerative Diseases

Neurodegenerative diseases like Alzheimer’s and Parkinson’s contribute to early death, and they represent a sizable slice of the overall mortality rate.28-30

Green tea extracts and EGCG fight the causes and progression of neurodegenerative diseases in multiple ways. For example, EGCG may prevent Alzheimer’s disease by reducing production of the toxic protein called beta-amyloid, as well as the resulting inflammation, which otherwise destroys brain cells.31,32

Not only does this increase the survival of brain cells, it also stimulates new brain cell production, especially in the memory-storage hippocampal area of the brain. These actions have resulted in improved behaviors in an animal model of Alzheimer’s.32-34



Cancer is the second-leading cause of death in the US, which makes reducing cancer risk an important way to reduce all-cause mortality.

Green tea polyphenols have been found to suppress cancer development and its aggressiveness in in vitro studies of many different cancers, including prostate, melanoma, breast, liver, lung, brain, and bladder malignancies.35-41

Epidemiological studies show that people who drink five or more cups of green tea per day have a 38% lower risk of developing liver cancer, compared with nondrinkers.38 In a group of men with prostate cancer, supplementation with green tea extract reduced markers of oxidative stress and increased natural protective mechanisms.39

Studies of breast cancer in relation to green tea consumption are particularly compelling, and deserve specific mention.

Green tea appears to have multiple effects on breast cancers, both before and after malignancies are discovered.

Studies show, for instance, that among women diagnosed with breast cancer, those with higher green tea consumption are less likely to have tumors spread to regional lymph nodes, or to metastasize, compared with women who consume little or none of the beverage.42

And green tea consumption correlates significantly with diminished recurrence rates of early breast cancers: consumption of three or more cups/day reduces recurrence risk by 31%, and among those with the very earliest (stage I) tumors, significant risk reduction of 57% has been observed.43

Even in studies not restricted to early-stage cancers, green tea consumption has shown a dose-related impact.

One large study showed that, compared with non-tea drinkers, those consuming, yearly, 250 or more grams of green tea leaves had risk reductions of 32% to 41% for breast cancer occurrence, figures generally supported in a large meta-analysis study as well.44,45

Lab studies shed additional light on green tea’s role in breast cancer prevention.

In culture experiments, EGCG was shown to inhibit rare but deadly inflammatory stem-like breast cancer cells, which contribute to extremely poor prognoses when found.46

And in living animals, green tea supplementation inhibited metastatic spread, and reduced the ability of metastases to grow in bones of mice with transplanted human breast tumor tissue.47

Finally, EGCG and green tea extracts are showing signs of effectiveness in the most challenging of breast cancer types, those lacking receptors for estrogen, in which relatively safe and simple treatment with estrogen-fighting drugs is not effective.48,49

Green tea extracts and EGCG act by a wide range of mechanisms to achieve these effects, including:

  • Inhibiting the out-of-control cell replication cycle typical of cancers,36,50
  • Inducing early cell death (apoptosis) in malignant cells,36,50
  • Suppressing the formation of new blood vessels to nourish rapidly-growing tumor masses, resulting in their destruction,35,51,52
  • Reducing production of the “protein-melting” enzymes tumors use to invade between cells and spread their malignant tissue,41,53,54
  • Inhibiting a host of proinflammatory signaling systems, including nuclear factor kappa B, and cyclooxygenase (COX).55

Oral Health

Oral health, tooth decay, and gum disease are rarely thought of as being associated with premature death, but that’s a dangerous misconception. The alarming truth is that people with gum disease can be anywhere from 34%-72% more likely to die of cardiovascular disease and other complications, compared to those in good oral health.56-59

Tooth loss has also been found to increase the risk of dying by up to 36% in older adults, while preventing tooth decay and loss can reduce that risk by 46%.60,61

Green tea extracts have been found to help slow or prevent tooth decay and loss. Research shows that green tea extract can reduce wear and roughness caused by chemical erosion on tooth material.62


Green Tea  

Studies published over the past decade are substantiating that green tea and its compounds can reduce our risk of dying from all causes.

Significant green tea-associated reductions in all-cause mortality have been reported in a long series of studies in multiple populations, with effects as modest as a 5% reduction, up to a 76% reduction in the risk of dying.1,7-11

These human longevity benefits correlate with the ability of green tea to protect cells throughout our aging bodies against degenerative alterations.

In response to higher intake of green tea and its extracts, studies reveal a reduced risk of developing cancer, diabetes, and vascular, neurodegenerative disorders.

Polyphenol extracts from green tea represent a low-cost addition to an overall strategy to prevent disease and extend life.

If you have any questions on the scientific content of this article, please call a Life Extension® Wellness Specialist at 1-866-864-3027.


  1. Liu J, Liu S, Zhou H, et al. Association of green tea consumption with mortality from all-cause, cardiovascular disease and cancer in a Chinese cohort of 165,000 adult men. Eur J Epidemiol. 2016;31(9):853-65.
  2. Babu PV, Liu D. Green tea catechins and cardiovascular health: an update. Curr Med Chem. 2008;15(18):1840-50.
  3. Suzuki Y, Miyoshi N, Isemura M. Health-promoting effects of green tea. Proc Jpn Acad Ser B Phys Biol Sci. 2012;88(3):88-101.
  4. Yang CS, Wang X. Green tea and cancer prevention. Nutr Cancer. 2010;62(7):931-7.
  5. Hursel R, Viechtbauer W, Westerterp-Plantenga MS. The effects of green tea on weight loss and weight maintenance: a meta-analysis. Int J Obes (Lond). 2009;33(9):956-61.
  6. Mousavi A, Vafa M, Neyestani T, et al. The effects of green tea consumption on metabolic and anthropometric indices in patients with Type 2 diabetes. J Res Med Sci. 2013;18(12):1080-6.
  7. Zhao LG, Li HL, Sun JW, et al. Green tea consumption and cause-specific mortality: Results from two prospective cohort studies in China. J Epidemiol. 2017;27(1):36-41.
  8. Kuriyama S, Shimazu T, Ohmori K, et al. Green tea consumption and mortality due to cardiovascular disease, cancer, and all causes in Japan: the Ohsaki study. JAMA. 2006;296(10):1255-65.
  9. Saito E, Inoue M, Sawada N, et al. Association of green tea consumption with mortality due to all causes and major causes of death in a Japanese population: the Japan Public Health Center-based Prospective Study (JPHC Study). Ann Epidemiol. 2015;25(7):512-8 e3.
  10. Suzuki E, Yorifuji T, Takao S, et al. Green tea consumption and mortality among Japanese elderly people: the prospective Shizuoka elderly cohort. Ann Epidemiol. 2009;19(10):732-9.
  11. Tang J, Zheng JS, Fang L, et al. Tea consumption and mortality of all cancers, CVD and all causes: a meta-analysis of eighteen prospective cohort studies. Br J Nutr. 2015;114(5):673-83.
  12. Arab H, Mahjoub S, Hajian-Tilaki K, et al. The effect of green tea consumption on oxidative stress markers and cognitive function in patients with Alzheimer’s disease: A prospective intervention study. Caspian J Intern Med. 2016;7(3):188-94.
  13. Babu PV, Sabitha KE, Shyamaladevi CS. Effect of green tea extract on advanced glycation and cross-linking of tail tendon collagen in streptozotocin induced diabetic rats. Food Chem Toxicol. 2008;46(1):280-5.
  14. Babu PV, Sabitha KE, Shyamaladevi CS. Therapeutic effect of green tea extract on advanced glycation and cross-linking of collagen in the aorta of streptozotocin diabetic rats. Clin Exp Pharmacol Physiol. 2006;33(4):351-7.
  15. British Society for Matrix Biology meeting. Manchester, 5-6 april 2004. Abstracts. Int J Exp Pathol. 2004;85(4):A45-77.
  16. Cavet ME, Harrington KL, Vollmer TR, et al. Anti-inflammatory and anti-oxidative effects of the green tea polyphenol epigallocatechin gallate in human corneal epithelial cells. Mol Vis. 2011;17:533-42.
  17. Dragicevic N, Smith A, Lin X, et al. Green tea epigallocatechin-3-gallate (EGCG) and other flavonoids reduce Alzheimer’s amyloid-induced mitochondrial dysfunction. J Alzheimers Dis. 2011;26(3):507-21.
  18. Jin JS, Touyama M, Hisada T, et al. Effects of green tea consumption on human fecal microbiota with special reference to Bifidobacterium species. Microbiol Immunol. 2012;56(11):729-39.
  19. Axling U, Olsson C, Xu J, et al. Green tea powder and Lactobacillus plantarum affect gut microbiota, lipid metabolism and inflammation in high-fat fed C57BL/6J mice. Nutr Metab (Lond). 2012;9(1):105.
  20. Liu Z, Chen Z, Guo H, et al. The modulatory effect of infusions of green tea, oolong tea, and black tea on gut microbiota in high-fat-induced obese mice. Food Funct. 2016;7(12):4869-79.
  21. Suzuki-Sugihara N, Kishimoto Y, Saita E, et al. Green tea catechins prevent low-density lipoprotein oxidation via their accumulation in low-density lipoprotein particles in humans. Nutr Res. 2016;36(1):16-23.
  22. Takechi R, Alfonso H, Hiramatsu N, et al. Elevated plasma and urinary concentrations of green tea catechins associated with improved plasma lipid profile in healthy Japanese women. Nutr Res. 2016;36(3):220-6.
  23. Yin J, Huang F, Yi Y, et al. EGCG attenuates atherosclerosis through the Jagged-1/Notch pathway. Int J Mol Med. 2016;37(2):398-406.
  24. Dulloo AG, Duret C, Rohrer D, et al. Efficacy of a green tea extract rich in catechin polyphenols and caffeine in increasing 24-h energy expenditure and fat oxidation in humans. Am J Clin Nutr. 1999;70(6):1040-5.
  25. Most J, van Can JG, van Dijk JW, et al. A 3-day EGCG-supplementation reduces interstitial lactate concentration in skeletal muscle of overweight subjects. Sci Rep. 2015;5:17896.
  26. Kumazoe M, Nakamura Y, Yamashita M, et al. Green Tea Polyphenol Epigallocatechin-3-gallate Suppresses Toll-like Receptor 4 Expression via Up-regulation of E3 Ubiquitin-protein Ligase RNF216. J Biol Chem. 2017;292(10):4077-88.
  27. Seo DB, Jeong HW, Kim YJ, et al. Fermented green tea extract exhibits hypolipidaemic effects through the inhibition of pancreatic lipase and promotion of energy expenditure. Br J Nutr. 2017;117(2):177-86.
  28. Desesquelles A, Demuru E, Salvatore MA, et al. Mortality from Alzheimer’s disease, Parkinson’s disease, and dementias in France and Italy: a comparison using the multiple cause-of-death approach. J Aging Health. 2014;26(2):283-315.
  29. Garcia-Ptacek S, Farahmand B, Kareholt I, et al. Mortality risk after dementia diagnosis by dementia type and underlying factors: a cohort of 15,209 patients based on the Swedish Dementia Registry. J Alzheimers Dis. 2014;41(2):467-77.
  30. Nabavi SF, Braidy N, Habtemariam S, et al. Neuroprotective Effects of Fisetin in Alzheimer’s and Parkinson’s Diseases: From Chemistry to Medicine. Curr Top Med Chem. 2016;16(17):1910-5.
  31. Rezai-Zadeh K, Shytle D, Sun N, et al. Green tea epigallocatechin-3-gallate (EGCG) modulates amyloid precursor protein cleavage and reduces cerebral amyloidosis in Alzheimer transgenic mice. J Neurosci. 2005;25(38):8807-14.
  32. Walker JM, Klakotskaia D, Ajit D, et al. Beneficial effects of dietary EGCG and voluntary exercise on behavior in an Alzheimer’s disease mouse model. J Alzheimers Dis. 2015;44(2):561-72.
  33. Ortiz-Lopez L, Marquez-Valadez B, Gomez-Sanchez A, et al. Green tea compound epigallo-catechin-3-gallate (EGCG) increases neuronal survival in adult hippocampal neurogenesis in vivo and in vitro. Neuroscience. 2016;322:208-20.
  34. Wang Y, Li M, Xu X, et al. Green tea epigallocatechin-3-gallate (EGCG) promotes neural progenitor cell proliferation and sonic hedgehog pathway activation during adult hippocampal neurogenesis. Mol Nutr Food Res. 2012;56(8):1292-303.
  35. Matsuo T, Miyata Y, Asai A, et al. Green Tea Polyphenol Induces Changes in Cancer-Related Factors in an Animal Model of Bladder Cancer. PLoS One. 2017;12(1):e0171091.
  36. Tsai YJ, Chen BH. Preparation of catechin extracts and nanoemulsions from green tea leaf waste and their inhibition effect on prostate cancer cell PC-3. Int J Nanomedicine. 2016;11:1907-26.
  37. Yamada S, Tsukamoto S, Huang Y, et al. Epigallocatechin-3-O-gallate up-regulates microRNA-let-7b expression by activating 67-kDa laminin receptor signaling in melanoma cells. Sci Rep. 2016;6:19225.
  38. Ni CX, Gong H, Liu Y, et al. Green Tea Consumption and the Risk of Liver Cancer: A Meta-Analysis. Nutr Cancer. 2017;69(2):211-20.
  39. Lassed S, Deus CM, Djebbari R, et al. Protective Effect of Green Tea (Camellia sinensis (L.) Kuntze) against Prostate Cancer: From In Vitro Data to Algerian Patients. Evid Based Complement Alternat Med. 2017;2017:1691568.
  40. Oya Y, Mondal A, Rawangkan A, et al. Down-regulation of histone deacetylase 4, -5 and -6 as a mechanism of synergistic enhancement of apoptosis in human lung cancer cells treated with the combination of a synthetic retinoid, Am80 and green tea catechin. J Nutr Biochem. 2017;42:7-16.
  41. Roomi MW, Kalinovsky T, Rath M, et al. Modulation of MMP-2 and MMP-9 secretion by cytokines, inducers and inhibitors in human glioblastoma T-98G cells. Oncol Rep. 2017;37(3):1907-13.
  42. Nakachi K, Suemasu K, Suga K, et al. Influence of drinking green tea on breast cancer malignancy among Japanese patients. Jpn J Cancer Res. 1998;89(3):254-61.
  43. Inoue M, Tajima K, Mizutani M, et al. Regular consumption of green tea and the risk of breast cancer recurrence: follow-up study from the Hospital-based Epidemiologic Research Program at Aichi Cancer Center (HERPACC), Japan. Cancer Lett. 2001;167(2):175-82.
  44. Zhang M, Holman CD, Huang JP, et al. Green tea and the prevention of breast cancer: a case-control study in Southeast China. Carcinogenesis. 2007;28(5):1074-8.
  45. Sun CL, Yuan JM, Koh WP, et al. Green tea, black tea and breast cancer risk: a meta-analysis of epidemiological studies. Carcinogenesis. 2006;27(7):1310-5.
  46. Mineva ND, Paulson KE, Naber SP, et al. Epigallocatechin-3-gallate inhibits stem-like inflammatory breast cancer cells. PLoS One. 2013;8(9):e73464.
  47. Luo KW, Ko CH, Yue GG, et al. Green tea (Camellia sinensis) extract inhibits both the metastasis and osteolytic components of mammary cancer 4T1 lesions in mice. J Nutr Biochem. 2014;25(4):395-403.
  48. Crew KD, Ho KA, Brown P, et al. Effects of a green tea extract, Polyphenon E, on systemic biomarkers of growth factor signalling in women with hormone receptor-negative breast cancer. J Hum Nutr Diet. 2015;28(3):272-82.
  49. Pan X, Zhao B, Song Z, et al. Estrogen receptor-alpha36 is involved in epigallocatechin-3-gallate induced growth inhibition of ER-negative breast cancer stem/progenitor cells. J Pharmacol Sci. 2016;130(2):85-93.
  50. Liu SM, Ou SY, Huang HH. Green tea polyphenols induce cell death in breast cancer MCF-7 cells through induction of cell cycle arrest and mitochondrial-mediated apoptosis. J Zhejiang Univ Sci B. 2017;18(2):89-98.
  51. Sartippour MR, Shao ZM, Heber D, et al. Green tea inhibits vascular endothelial growth factor (VEGF) induction in human breast cancer cells. J Nutr. 2002;132(8):2307-11.
  52. Gu JW, Makey KL, Tucker KB, et al. EGCG, a major green tea catechin suppresses breast tumor angiogenesis and growth via inhibiting the activation of HIF-1alpha and NFkappaB, and VEGF expression. Vasc Cell. 2013;5(1):9.
  53. Annabi B, Lachambre MP, Bousquet-Gagnon N, et al. Green tea polyphenol (-)-epigallocatechin 3-gallate inhibits MMP-2 secretion and MT1-MMP-driven migration in glioblastoma cells. Biochim Biophys Acta. 2002;1542(1-3):209-20.
  54. Deb G, Thakur VS, Limaye AM, et al. Epigenetic induction of tissue inhibitor of matrix metalloproteinase-3 by green tea polyphenols in breast cancer cells. Mol Carcinog. 2015;54(6):485-99.
  55. Roy P, George J, Srivastava S, et al. Inhibitory effects of tea polyphenols by targeting cyclooxygenase-2 through regulation of nuclear factor kappa B, Akt and p53 in rat mammary tumors. Invest New Drugs. 2011;29(2):225-31.
  56. Blaizot A, Vergnes JN, Nuwwareh S, et al. Periodontal diseases and cardiovascular events: meta-analysis of observational studies. Int Dent J. 2009;59(4):197-209.
  57. DeStefano F, Anda RF, Kahn HS, et al. Dental disease and risk of coronary heart disease and mortality. BMJ. 1993;306(6879):688-91.
  58. Dorn JM, Genco RJ, Grossi SG, et al. Periodontal disease and recurrent cardiovascular events in survivors of myocardial infarction (MI): the Western New York Acute MI Study. J Periodontol. 2010;81(4):502-11.
  59. Hung HC, Joshipura KJ, Colditz G, et al. The association between tooth loss and coronary heart disease in men and women. J Public Health Dent. 2004;64(4):209-15.
  60. Hayasaka K, Tomata Y, Aida J, et al. Tooth loss and mortality in elderly Japanese adults: effect of oral care. J Am Geriatr Soc. 2013;61(5):815-20.
  61. Hu HY, Lee YL, Lin SY, et al. Association Between Tooth Loss, Body Mass Index, and All-Cause Mortality Among Elderly Patients in Taiwan. Medicine (Baltimore). 2015;94(39):e1543.
  62. De Moraes MD, Carneiro JR, Passos VF, et al. Effect of green tea as a protective measure against dental erosion in coronary dentine. Braz Oral Res. 2016;30.